“Eating sugar makes children hyperactive.”

The idea that a sugar rush sends children bouncing off the walls is a persistent belief, but its roots can be traced back to the 1970s. During this time, a pediatric allergist named Ben Feingold published a best-selling book, "Why Your Child Is Hyperactive," which, despite lacking substantial evidence, suggested a link between food additives, including sugar, and excitable behavior in children. This widely read book played a significant role in popularizing the notion of a direct causal link between sugar and hyperactivity.

However, extensive scientific research conducted since then has consistently debunked this claim. Numerous thorough reviews, including double-blind studies where neither parents nor researchers knew if children were consuming sugar or a placebo, have found no direct association between sugar intake and hyperactivity in children. Experts, such as the chair of the American Academy of Pediatrics Committee on Nutrition, Mark Corkins, confirm that there is an overwhelming consensus among researchers that no such link exists. The Centers for Disease Control and Prevention also states that research does not support the view that eating too much sugar causes hyperactivity.

Despite the scientific evidence, the myth endures largely due to perception and association. Parents often observe children becoming more energetic at events like birthday parties or holidays, which are typically rich in sugary treats. In these celebratory environments, the excitement of the situation, the presence of friends (Review) and relatives, and the general stimulation are far more likely to contribute to heightened activity levels than the sugar itself. Furthermore, parental expectations can play a role; studies have shown that parents who believe sugar affects behavior are more likely to perceive their children as hyperactive after they think their child has consumed sugar, even if they haven't. While limiting excessive sugar intake is important for overall health, it does not directly cause hyperactivity.